A Cardiac Patient remains a Cardiac Patient for life. Why?

One of the questions I hear most often from cardiac patients is some version of: “I had the surgery, I’m on my medications, so why don’t I feel like myself again?” Rajesh’s story answers that question better than I ever could in a consultation room.

– Dr Anupam Gaur

The Story of Rajesh Verma

RV
Rajesh Verma
Senior Executive, 58 · Delhi NCR
Post-angioplasty recovery · Cardiac bioterrain programme
2019 — Heart attack at 54
Emergency angioplasty. Two stents placed. Discharged with medications for life: aspirin, statins, beta-blockers. Told I was “fixed.”
2022 — Discovered the missed signals
Dr. Gaur reviewed my echocardiography reports going back to 2015. Grade 1 diastolic dysfunction had been documented: four years before my heart attack. Nobody flagged it. Nobody acted on it.
2024 — Today
Energy levels I haven’t felt since my 40s. Biomarkers all within optimal range. My cardiologist’s words at my last review: “I don’t know what you’re doing, but keep doing it.”

I had what the system would call a successful outcome. Emergency care was excellent; fast, precise, life-saving. The stents did their job. But two years after my heart attack, I still felt like a cardiac patient. I was breathless climbing stairs. Fatigue by 3 pm. Living in fear of the next episode.

A colleague referred me to Dr. Gaur. What he showed me in our first consultation stopped me cold.

“Your 2015 echocardiogram noted Grade 1 diastolic dysfunction. Your 2017 report noted the same: slightly worse. Your 2018 annual checkup: again, documented, again unremarked upon. Your heart was sending signals for four years. The signal was in the reports. No one connected it to what was coming.”

It was not anyone’s fault. My cardiologist was not negligent. He was doing exactly what his training and the system expected of him — managing the event. What was missing was someone looking at the terrain that produced the event. That is an entirely different job. And it is the job Dr. Gaur does.

He explained that diastolic dysfunction: the heart’s inability to relax properly, is an early signal of microvascular inflammation, metabolic stress, and myocardial stiffening. My fatty liver, my VAT/SAT ratio, my quietly rising hs-CRP over those years, all of it was readable as a trajectory. Nobody read it as one.

Over 18 months, working alongside my cardiologist (this is important – Dr. Gaur never asked me to stop any prescribed medication), we addressed the underlying terrain. Mitochondrial support. Metabolic rehabilitation. Anti-inflammatory nutrition protocols. A structured approach to reversing the microvascular damage. Serial testing showed consistent improvement at each stage.

Today, at 58, I feel… and my tests confirm, better than I did at 52. My diastolic function has normalised. My inflammatory markers are in the lowest-risk band. I have more energy than I had in the years leading up to my heart attack. If I am honest, those years of fatigue and breathlessness were themselves a signal I had normalised into my routine.

I think about the version of events where someone had read those 2015 reports differently. Where the diastolic dysfunction had been seen not as an isolated cardiac finding, but as evidence of a terrain that needed attention. I might never have needed the stents at all.

That conversation cannot happen in a 10-minute consultation with a specialist managing a thousand patients. But it can happen at CFOH. And for me, it changed everything.